The Red Raiders celebrate after defeating Mainland last week in the South Jersey Group 4 playoffs. (Photos courtesy of OCHSfootball.com) By TIM KELLYGoogle Maps says Long Branch High School is 85.9 miles from Ocean City. The geographical divide is the most obvious thing separating the Ocean City High football team from its opponent in Friday night’s playoff game, but certainly not the only thing.“I’ll be honest, I don’t know much at all about Long Branch,” Red Raider Head Coach Kevin Smith said following his team’s thrilling 21-14 win in their opening round playoff game at Mainland last Saturday.Of course Long Branch has the same issue in preparing to face O.C.Though Smith has undoubtedly immersed himself in all things Green Wave since then, the Red Raider coach had more pressing matters to deal with at the time, namely directing his team at Mainland. It was decided in the final seconds and avenged a bitter 21-6 loss to the Mustangs in a regular season game the week before.With the win, O.C. earned the right to travel up the Garden State Parkway for a 7 p.m. Friday date at Long Branch (4-5), the sixth-seeded team in the tournament.The last time Ocean City won a playoff game it was 2001, a 21-14 victory over Lacey.“You just added to the storied tradition of the Ocean City-Mainland rivalry,” Smith told his seventh-seeded team after they used a goal-line stand and fumble recovery to knock off their archrivals.Mainland came into the playoffs as the undefeated and untied No. 2 seed.“I couldn’t be more proud of our kids for how hard they played, and for not getting discouraged when Mainland scored first,” Smith said.Smith would be the first to say that’s all history now and fodder for off-season chatter. The nature of the state playoffs doesn’t allow for relaxation, much less celebration. It does cause one to wonder why Long Branch, located in the northern reaches of Monmouth County, is part of the South Jersey Group 4 playoffs.Ocean City dominated the line of scrimmage against Mainland.Why must Ocean City travel closer to New York City than to Philadelphia in order to play a team in the “South” section?Jack DuBoise, assistant director of the New Jersey State Interscholastic Athletic Association, said it was part of a new pairing system in which the top seeds of natural geographic regions stay in their normal areas, and the second and third seeds are sent to the opposite region. The next two seeds stay in their home environs and the fifth and sixth seeds are moved.The process, known as “snaking” the brackets, is designed to create a fairer seeding system for all the playoff teams and to avoid blowouts and mismatches, DuBoise explained. However, it comes at a price of sometimes breaking up the traditional regions, as was the case for Ocean City this year.“I think (the snaking process) has worked out well and it has created very interesting matchups,” DuBoise said when reached on Wednesday.Some Ocean City working families, faced with the prospect of trying to arrive in Long Branch by 7 p.m. on a Friday, would probably take issue with that assessment. Yet the fact remains, the Raiders have advanced in the big dance and can make the South Jersey final for the first time since 2000 with a win.Long Branch knocked off third-seeded Highland, 33-3, due at least in part to the fact Highland was missing 17 players for disciplinary reasons. The Green Wave won the last two Central Jersey Group 4 titles prior to being “snaked” to the South region, and they showed their playoff mettle against Highland.Long Branch also has a deceptive record. Even though they are just 4-5, every opponent on the Wave’s schedule is a playoff team.If the Red Raiders can prevail against Long Branch, they would next be matched against the winner of the other semi-final pitting Millville at Shawnee, the two-time defending champions.Ocean City’s Chris Armstrong, right, exults with Brian Beckmann (6) after Armstrong’s game-clinching fumble recovery at Mainland.
Martin Lightbody has stepped down as chief executive of Finsbury Foods, with immediate effect, as the Group announces an improved second-half performance in its full-year results. Lightbody will be replaced by chief operating officer John Duffy and will assume the position of non-executive chairman following the firm’s Annual General Meeting on 25 November.Lightbody told British Baker it had always been the plan to hold the position of chief executive for a 12-month period, following the departure of former CEO Dave Brooks last year, but said that he would still be “heavily involved with the business” in his new role.Finsbury’s results for the 53 weeks to 4 July 2009 revealed revenue growth of 8%, with like-for-like sales up 2%. However, it said profits had suffered with the high cost of raw materials such as eggs, chocolate and sugar.Group revenue stood at £178.9m (52 week period to 28 June 2008: £165.1m), an increase of £13.8m (8.4%) year-on-year. Adjusted profit before tax was £5.0m (2008: £7.7m).However the firm announced a 78% “improvement” in adjusted profit before tax in the second half of the year, compared to the first.Its bread and ‘free from’ division saw like-for-like growth up 14%, and larger cake sales continued to grow in line with the overall ambient cake market, up 2% on last year.Finsbury has seen sales of its Thorntons branded cakes rocket by over 70% during the year, making it the fastest-growing brand in the cake market, according to the firm. Sales of its WeightWatchers branded cakes also rose, up 25%.Duffy has assisted Lightbody since September 2008, and has held previous positions at WT Foods, Noon Products, Golden Wonder and Mars.To read the full story see the next issue of British Baker, out 9 October.
Limitations in the Commission’s powers at the time meant it was not able to disqualify trustees B and C because their trusteeships had lapsed by this point. The Commission successfully fought for new powers to address this, which were granted under the Charities (Protection and Social Investment) Act 2016, putting the Commission in a stronger position to disrupt and stop the abuse and mismanagement of charities.The Commission worked closely with the interim manager and newly appointed trustees to ensure that the recovered and remaining funds could be put to good use. The new trustees selected Nottingham Miners Welfare Trust Scheme (NMWTFS) as their preferred recipient charity. Following due diligence checks by the inquiry £1,142,573 was transferred to NMWTFS to support people connected with the Nottinghamshire area coal field, including vulnerable beneficiaries.NMH was wound up and removed from the register of charities in January 2017. A full report of the inquiry is available on GOV.UK.Ends.Notes to Editors The inquiry highlights risks that can arise from charities being closely linked to a non-charitable organisation. No charity should ever use or be used by non-charitable organisations to pursue uncharitable interests. Newly published guidance from the Commission helps equip trustees to manage these types of risks. The guidance is available on GOV.UK *As the term of Trustee B’s trusteeship had lapsed under the provisions of the charity’s governing document, it was not necessary for the inquiry to take action to remove him. Email [email protected] Press office Press mobile – out of hours only 07785 748787 Trustees that benefitted from the use of charity funds for private building works were guilty of serious misconduct and mismanagement in the running of the charity, a Charity Commission inquiry has found. A trustee is now disqualified and was ordered to repay funds, resulting in over £200,000 being recovered.The regulator opened a statutory inquiry into Nottinghamshire Miners Home in August 2007 after the Serious Fraud Office (SFO) raised concerns that the charity’s trading subsidiary, Phoenix Nursing and Residential Home Ltd (PNRHL), may be being misused for the private benefit of trustees A and B and their families. This also brought up wider concerns about Trustees A, B and C’s management and oversight of the charity.The inquiry liaised closely with the SFO and South Yorkshire Police. Commission investigators used powers to obtain information from the banks of NMH, PNRHL, the trustees and the charity’s accountant, which revealed that trustees A and B benefitted from £150,000 of charitable funds, through fraudulent invoicing, which were spent on building works carried out at private properties connected to the two trustees.The Commission provided witness statements to support the prosecution, and gave evidence at the hearing in 2012, which resulted in trustee A being convicted of 14 counts of theft and automatically disqualified from trusteeship. Trustee B was found not guilty, however the Commission maintained that they were responsible for misconduct and/or mismanagement.*A claim was made under the Proceeds of Crime Act, and Trustee A was ordered to pay over £200,000 compensation to the charity, including over £50,000 in interest.Protective actionAt the time of opening the inquiry, the charity had informed the Commission that it intended to sell a care home in Lincolnshire which it leased to PNRHL, claiming that it was failing as a result of the decline of the mining industry. Concerned that c. £1.5 million in sale proceeds could be at risk, the inquiry placed legal restrictions on the charity’s bank accounts, those of its trading subsidiary and on the charity’s solicitor. Both trustees A and B were suspended from their roles, and the Commission appointed an interim manager to take over the management of the charity in 2008.Serious misconduct and mismanagementFollowing interviews and further examination of evidence, the inquiry concluded that all three trustees failed to adequately discharge their legal duties as trustees. They also failed in their responsibility to review the performance of the trading subsidiary in administering the care home.Trustee A and B’s use of charitable funds for private building works and unauthorised personal benefit clearly amounted to serious misconduct and mismanagement in the administration of the charity.Harvey Grenville, Head of Investigation and Enforcement at the Charity Commission said: This case involved an appalling and cynical misuse of funds intended for deserving people. Through the diversion of money for personal comfort, vital resources were taken from those they were there to help. Charities exist to improve lives and strengthen society, but the actions of these individuals meant that a community was badly let down. Our intervention provided vital support to prosecutors, ensured that those responsible faced the consequences of their actions and enabled the sizeable recovery of charity funds. This should send a strong signal that this type of abuse will not be tolerated. This inquiry protected important charitable assets and ensured that funds could be put to good use for mining communities in Nottinghamshire.
Related By phone and online, the care continues Global race to a COVID-19 vaccine This is part of our Coronavirus Update series in which Harvard specialists in epidemiology, infectious disease, economics, politics, and other disciplines offer insights into what the latest developments in the COVID-19 outbreak may bring.Lung injury and acute respiratory distress syndrome have taken center stage as the most dreaded complications of COVID-19, the disease caused by the new coronavirus, SARS-CoV-2. But heart damage has recently emerged as yet another grim outcome in the virus’s repertoire of possible complications.COVID-19 is a spectrum disease, spanning the gamut from barely symptomatic infection to critical illness. Reassuringly, for the large majority of individuals infected with the new coronavirus, the ailment remains in the mild-to-moderate range.Yet, a number of those infected develop heart-related problems either out of the blue or as a complication of preexisting cardiac disease. A report from the early days of the epidemic described the extent of cardiac injury among 41 patients hospitalized with COVID-19 in Wuhan, China: Five, or 12 percent, had signs of cardiovascular damage. These patients had both elevated levels of cardiac troponin — a protein released in the blood by the injured heart muscle — and abnormalities on electrocardiograms and heart ultrasounds. Since then, other reports have affirmed that cardiac injury can be part of coronavirus-induced harm. Moreover, some reports detail clinical scenarios in which patients’ initial symptoms were cardiovascular rather than respiratory in nature. How does the new coronavirus stoke cardiac damage?The ways in which the new coronavirus provokes cardiac injury are neither that new nor surprising, according to Harvard Medical School physician-scientists Peter Libby and Paul Ridker. The part that remains unclear is whether SARS-CoV-2 is somehow more virulent toward the heart than other viruses. Libby and Ridker, who are practicing cardiologists at Brigham and Women’s, say COVID-19-related heart injury could occur in any several ways.First, people with preexisting heart disease are at a greater risk for severe cardiovascular and respiratory complications from COVID-19. Similarly, research has shown that infection with the influenza virus poses a more severe threat for people with heart disease than those without cardiac problems. Research also shows that heart attacks can actually be brought on by respiratory infections such as the flu. Second, people with previously undiagnosed heart disease may be presenting with previously silent cardiac symptoms unmasked by the viral infection. In people with existing heart-vessel blockages, infection, fever, and inflammation can destabilize previously asymptomatic fatty plaques inside the heart vessels. Fever and inflammation also render the blood more prone to clotting, while also interfering with the body’s ability to dissolve clots — a one-two punch akin to throwing gasoline on smoldering embers.“It’s like one big stress test for the heart,” said Ridker, the Eugene Braunwald Professor of Medicine at Brigham and Women’s Hospital.,Third, some people may experience heart damage that mimics heart attack injury even if their arteries lack the fatty, calcified flow-limiting blockages known to cause classic heart attacks. This scenario can occur when the heart muscle is starved for oxygen, which in the case of COVID-19 may be triggered by a mismatch between oxygen supply and oxygen demand. Fever and inflammation accelerate heart rate and increase metabolic demands on many organs, including the heart. That stress is compounded if the lungs are infected and incapable of exchanging oxygen and carbon dioxide optimally. This impaired gas exchange can further diminish oxygen supply to the heart muscle.Finally, there is a subset of people with COVID-19 — some of them previously healthy and with no underlying cardiac problems — who develop fulminant inflammation of the heart muscle as a result of the virus directly infecting the heart. This type of inflammation could lead to heart rhythm disturbances and cardiac muscle damage as well as interfere with the heart’s ability to pump blood optimally. The propensity of certain viruses to attack the heart muscle and cause viral myocarditis is well known, Libby said, adding that the most notorious viral offender has been the Coxsackie B virus. A recent case report from Italy underscores the notion that the new coronavirus could also infect the heart and affect heart muscle function in healthy adults even after the acute phase of the infection has resolved and even in the absence of lung damage.“There are definitely some people who develop acute fulminant myocarditis — in which the virus infects the heart muscle itself or the cells within the heart — and causes a horrible inflammatory reaction,” said Libby, the Mallinckrodt Professor of Medicine at Brigham and Women’s Hospital. “This can be life threatening, and it can happen in people who don’t have any preexisting risk factors.”Libby and Ridker, however, say this out-of-the-blue scenario in otherwise healthy individuals is likely rare relative to the overall number of people with COVID-19 who experience heart problems. The frenemy withinFor Ridker and Libby, the cardiac involvement in COVID-19 is yet another striking example of the widespread effects of inflammation on multiple organs and systems. Inflammation is a critical defense response during infection, but it has a dark side. Infections can set off a cascade of immune signals that affect various organs. Libby and Ridker hypothesize that any infection in the body — a festering boil, an injured joint, a virus — can become a source of inflammation that activates the release of inflammatory proteins known as cytokines and calls up armies of white blood cells and other messenger molecules that, in an effort to fight the infection, disrupt normal processes. When these inflammatory molecules reach the welcoming soil of a fatty deposit in the blood vessel wall — one that is already studded with resident inflammatory white blood cells — the cytokines can boost the local inflammatory response and trigger a heart attack.“Our work has shown that cytokines can impinge on these cells in the plaque and push it through a round of further activation,” Libby said. “[Heart inflammation] can be life threatening, and it can happen in people who don’t have any preexisting risk factors.” — Peter Libby The inflammatory chemicals released during infection can also induce the liver to ramp up the production of important proteins that defend the body from infection. These proteins, however, make the blood more prone to clotting, while also reducing the secretion of natural clot-dissolving substances. The tiny clots that may form can clog the small blood vessels in the heart and other organs, such as the kidneys, depriving them of oxygen and nutrients and setting the stage for the multisystem failure that can occur in acute infection.Thus, immune-mediated injury to the heart and other organs could be collateral damage because of the body’s overwhelming systemic immune response — a condition known as cytokine storm, which is marked by the widespread release of cytokines that can cause cellular demise, tissue injury and organ damage.COVID-19 and blood pressure medicationsSARS-CoV-2 invades human cells by latching its spike protein onto the ACE2 receptor found on the surface of cells in the airways, lungs, heart, kidneys and blood vessels. The ACE2 protein is an important player in the renin-angiotensin-aldosterone system, which regulates blood vessel dilation and blood pressure. Two classes of drugs widely used to treat high blood pressure and heart disease — ACE inhibitors and angiotensin receptor blockers — interact with the ACE2 receptor. A possible concern related to COVID-19 stems from the notion that these blood pressure medications could increase the number of ACE2 receptors expressed on cells, possibly creating more molecular gates for the virus to enter. Some experts have wondered whether the use of such drugs could render people who take them more susceptible to infection. Conversely, others have postulated that the abundance of ACE2 receptors may enhance cardiovascular function, exercising a protective effect during infection.The answer is far from clear, but a recent review suggests these medicines may play a dual role in COVID-19 — on the one hand, enhancing susceptibility to infection and, on the other, protecting the heart and ameliorating lung damage from the disease.Libby and Ridker cautioned that patients who take such life-saving medications should stay on them or at least have a careful discussion with their cardiologists. This is because these drugs have clear and well-established benefits in hypertension and certain forms of heart disease, while their propensity to make humans more susceptible to SARS-CoV-2 remains speculative for the time being.But what remains speculative today will crystalize in the weeks and months to come, Ridker and Libby said, because the science is moving forward rapidly, with new papers coming out daily and a growing pool of patients to draw observations from. Team at Harvard plans to launch clinical trial in fall Health Services director Giang Nguyen talks about adding remote services and new resources for Harvard community Hope for managing hospital admissions of COVID-19 cases “In 12 to 18 months we’re going to have a great deal of information, but right now our job is to, number one, keep people from getting COVID-19 by strict adherence to now-familiar containment measures,” Libby said. “Then, we need to get people who get the disease through this acute phase.”The need for rigorous randomized trials done quickly and effectively is acute, they said. Until the evidence from these trials begins to coalesce, clinicians will have to navigate the uncharted territory of delivering cardiac care in the time of pandemic with caution but also with resolve.“We don’t have the comfort of our usual databases, so we have to rely on our clinical skills and judgment. But we have to do so in all humility because often data don’t bear out our logical preconceptions,” Libby said. “Yet, we must act.” New projections suggest social-distancing measures in state may be flattening the curve
Officials have arrested a 52-year-old Lake Worth man who reportedly molested a teen after luring the teen into his work truck with an offer to pay him cash to help do yard work.The incident occurred on on July 6th as the teen and his friend were walking towards N Street in Lake Worth.According to the report, Osvaldo Vicente approached the two teens in what appeared to be a landscaper’s work truck and offered the teens money to help cut grass and pick up trash at a home a block away from where they were walking.The two teens agreed, however, that’s when Vicente reportedly told the teens that he could only take one of them. The oldest of the teens agreed to go with him and jumped in the vehicle.Vicentre began driving however, the teen became alarmed with the situation when he noticed that they were headed towards Interstate 95. The teen then asked where they were headed and that’s when Vicentre reportedly began smooth-talking the teen and touching him inappropriately. Vicentre drove south on Interstate 95 towards an exit in Delray. Once the vehicle stopped the teen jumped out and ran to a gas station for help.Vicentre was eventually located on Saturday arrested.He is now facing kidnapping and child molestation charges.Authorities believe that Vicentre may have had more Victims and are asking for anyone who believes they may have been a victim to contact Crime Stoppers at 800-458-TIPS.