May 12, 2021 Find out more March 28, 2007 – Updated on January 20, 2016 Sentence called for in Gaddafi libel trial condemned as outrageous News Algeria : Reporter jailed after covering Tuareg protests in southern Algeria RSF_en Help by sharing this information Reporters Without Borders is dismayed by the heavy sentence demanded on appeal against Ali Fodil and Naïla Berrahal, respectively managing editor and journalist on the Arabic-language daily Ech-Chourouk, for allegedly defaming Libyan President Muammar Gaddafi.The prosecutor at the Algiers appeal court on 21 March called for a one-year prison sentence and 500,000 dinars (5,300 euros) fine against the two journalists along with a one-year ban on the newspaper.The libel suit against the two journalists was taken out in the name of the Libyan leader at the start of October 2006, by the Libyan representative in Algiers. It cited two articles, carried by the newspaper in August which referred to tensions in the region having been raised by Gaddafi’s involvement in discussions with the Tuareg community in Algeria.The Libyan leader challenged the truth of the articles, which he said had personally “damaged” him, the Libyan state and the security of both Algeria and Libya.A lower court in Hussein-Dey in the eastern Algiers suburbs sentenced the journalists on 31 October 2006 to six months in prison, fines of 20,000 dinars (220 euros) and 500,000 dinars (5,300 euros) in damages. The judge based the sentence on Article 144a of the criminal code relating to defamation of a head of state, an offence punishable by 12 months in prison. The court also ordered a two-month closure of the daily.Ali Fodil and Naïla Berrahal appealed against the sentence, which their lawyer, Khaled Bergheul, described as “very harsh in relation to the facts”. Sentence was adjourned, allowing the journalists to remain free while awaiting the appeal. The court in Algiers is due to hand down its sentence on 4 April.“Algerian journalists work without protection. The terms of the press law, which the authorities refuse to amend, are vague and capable of being interpreted according to the decision of individual judges,” the worldwide press freedom organisation said. “Journalists are not treated fairly by the courts, which continue to serve the interests of the government,” it concluded. May 18, 2021 Find out more Follow the news on Algeria Receive email alerts to go further News AlgeriaMiddle East – North Africa AlgeriaMiddle East – North Africa News Algeria pressures reporters by delaying renewal of accreditation News Harassment of Algerian reporters intensifies in run-up to parliamentary elections April 29, 2021 Find out more Organisation
COIS Fharraige has been brought to a close for another year. Kilkee was treated to over an hour of the Zutons biggest hits as they finished out the musical proceedings. The biggest cheer of the weekend came when the band launched into Valerie mid setLimerick jazz ensemble Jazz Lite played homage to the Beatles with a fab four medley during their set in the afternoon. The Lightning Seeds gave a special acoustic performance while The Stereo MCs sent the crowd wild when they performed their 90’s hit Connected.Sign up for the weekly Limerick Post newsletter Sign Up Saturday’s headliners The Saw Doctors did what they do best – getting the crowd into the party mood kicking off their set with N17 and sending the crowd home delighted after playing hit after hit. With the weather reaching soaring heights over the weekend, Cois Fharraige patrons gathered in numbers to enjoy the many beach based activities running alongside the music including three time Irish Wind Surfing champion Ryan Coote who demonstrated his windsurfing talents to the Kilkee crowd.The weekend also saw a successful return of the famed Tivoli Cup Racquets tournament. An emblem of a long-running competition that is played against the plastered white walls on Kilkee beach, the game is unique to the seaside town and a favourite of hell raising film star Richard Harris who holds the title for most consecutive victories from 1948-51. This year’s winners were Trevor Whelan and Ronan McDonagh from Limerick and were presented as champions by the Mayor of Kilkee Lily Marrinan Sullivan. Print Facebook Twitter Linkedin WhatsApp NewsLocal NewsCois Fharraige and weather reaches soaring heightsBy admin – September 14, 2009 578 Advertisement Email Previous articleAer Lingus will not use Shannon Pre-Clearance Facilities before 2010Next articleLocal economy in recovery mode admin
Related By phone and online, the care continues Global race to a COVID-19 vaccine This is part of our Coronavirus Update series in which Harvard specialists in epidemiology, infectious disease, economics, politics, and other disciplines offer insights into what the latest developments in the COVID-19 outbreak may bring.Lung injury and acute respiratory distress syndrome have taken center stage as the most dreaded complications of COVID-19, the disease caused by the new coronavirus, SARS-CoV-2. But heart damage has recently emerged as yet another grim outcome in the virus’s repertoire of possible complications.COVID-19 is a spectrum disease, spanning the gamut from barely symptomatic infection to critical illness. Reassuringly, for the large majority of individuals infected with the new coronavirus, the ailment remains in the mild-to-moderate range.Yet, a number of those infected develop heart-related problems either out of the blue or as a complication of preexisting cardiac disease. A report from the early days of the epidemic described the extent of cardiac injury among 41 patients hospitalized with COVID-19 in Wuhan, China: Five, or 12 percent, had signs of cardiovascular damage. These patients had both elevated levels of cardiac troponin — a protein released in the blood by the injured heart muscle — and abnormalities on electrocardiograms and heart ultrasounds. Since then, other reports have affirmed that cardiac injury can be part of coronavirus-induced harm. Moreover, some reports detail clinical scenarios in which patients’ initial symptoms were cardiovascular rather than respiratory in nature. How does the new coronavirus stoke cardiac damage?The ways in which the new coronavirus provokes cardiac injury are neither that new nor surprising, according to Harvard Medical School physician-scientists Peter Libby and Paul Ridker. The part that remains unclear is whether SARS-CoV-2 is somehow more virulent toward the heart than other viruses. Libby and Ridker, who are practicing cardiologists at Brigham and Women’s, say COVID-19-related heart injury could occur in any several ways.First, people with preexisting heart disease are at a greater risk for severe cardiovascular and respiratory complications from COVID-19. Similarly, research has shown that infection with the influenza virus poses a more severe threat for people with heart disease than those without cardiac problems. Research also shows that heart attacks can actually be brought on by respiratory infections such as the flu. Second, people with previously undiagnosed heart disease may be presenting with previously silent cardiac symptoms unmasked by the viral infection. In people with existing heart-vessel blockages, infection, fever, and inflammation can destabilize previously asymptomatic fatty plaques inside the heart vessels. Fever and inflammation also render the blood more prone to clotting, while also interfering with the body’s ability to dissolve clots — a one-two punch akin to throwing gasoline on smoldering embers.“It’s like one big stress test for the heart,” said Ridker, the Eugene Braunwald Professor of Medicine at Brigham and Women’s Hospital.,Third, some people may experience heart damage that mimics heart attack injury even if their arteries lack the fatty, calcified flow-limiting blockages known to cause classic heart attacks. This scenario can occur when the heart muscle is starved for oxygen, which in the case of COVID-19 may be triggered by a mismatch between oxygen supply and oxygen demand. Fever and inflammation accelerate heart rate and increase metabolic demands on many organs, including the heart. That stress is compounded if the lungs are infected and incapable of exchanging oxygen and carbon dioxide optimally. This impaired gas exchange can further diminish oxygen supply to the heart muscle.Finally, there is a subset of people with COVID-19 — some of them previously healthy and with no underlying cardiac problems — who develop fulminant inflammation of the heart muscle as a result of the virus directly infecting the heart. This type of inflammation could lead to heart rhythm disturbances and cardiac muscle damage as well as interfere with the heart’s ability to pump blood optimally. The propensity of certain viruses to attack the heart muscle and cause viral myocarditis is well known, Libby said, adding that the most notorious viral offender has been the Coxsackie B virus. A recent case report from Italy underscores the notion that the new coronavirus could also infect the heart and affect heart muscle function in healthy adults even after the acute phase of the infection has resolved and even in the absence of lung damage.“There are definitely some people who develop acute fulminant myocarditis — in which the virus infects the heart muscle itself or the cells within the heart — and causes a horrible inflammatory reaction,” said Libby, the Mallinckrodt Professor of Medicine at Brigham and Women’s Hospital. “This can be life threatening, and it can happen in people who don’t have any preexisting risk factors.”Libby and Ridker, however, say this out-of-the-blue scenario in otherwise healthy individuals is likely rare relative to the overall number of people with COVID-19 who experience heart problems. The frenemy withinFor Ridker and Libby, the cardiac involvement in COVID-19 is yet another striking example of the widespread effects of inflammation on multiple organs and systems. Inflammation is a critical defense response during infection, but it has a dark side. Infections can set off a cascade of immune signals that affect various organs. Libby and Ridker hypothesize that any infection in the body — a festering boil, an injured joint, a virus — can become a source of inflammation that activates the release of inflammatory proteins known as cytokines and calls up armies of white blood cells and other messenger molecules that, in an effort to fight the infection, disrupt normal processes. When these inflammatory molecules reach the welcoming soil of a fatty deposit in the blood vessel wall — one that is already studded with resident inflammatory white blood cells — the cytokines can boost the local inflammatory response and trigger a heart attack.“Our work has shown that cytokines can impinge on these cells in the plaque and push it through a round of further activation,” Libby said. “[Heart inflammation] can be life threatening, and it can happen in people who don’t have any preexisting risk factors.” — Peter Libby The inflammatory chemicals released during infection can also induce the liver to ramp up the production of important proteins that defend the body from infection. These proteins, however, make the blood more prone to clotting, while also reducing the secretion of natural clot-dissolving substances. The tiny clots that may form can clog the small blood vessels in the heart and other organs, such as the kidneys, depriving them of oxygen and nutrients and setting the stage for the multisystem failure that can occur in acute infection.Thus, immune-mediated injury to the heart and other organs could be collateral damage because of the body’s overwhelming systemic immune response — a condition known as cytokine storm, which is marked by the widespread release of cytokines that can cause cellular demise, tissue injury and organ damage.COVID-19 and blood pressure medicationsSARS-CoV-2 invades human cells by latching its spike protein onto the ACE2 receptor found on the surface of cells in the airways, lungs, heart, kidneys and blood vessels. The ACE2 protein is an important player in the renin-angiotensin-aldosterone system, which regulates blood vessel dilation and blood pressure. Two classes of drugs widely used to treat high blood pressure and heart disease — ACE inhibitors and angiotensin receptor blockers — interact with the ACE2 receptor. A possible concern related to COVID-19 stems from the notion that these blood pressure medications could increase the number of ACE2 receptors expressed on cells, possibly creating more molecular gates for the virus to enter. Some experts have wondered whether the use of such drugs could render people who take them more susceptible to infection. Conversely, others have postulated that the abundance of ACE2 receptors may enhance cardiovascular function, exercising a protective effect during infection.The answer is far from clear, but a recent review suggests these medicines may play a dual role in COVID-19 — on the one hand, enhancing susceptibility to infection and, on the other, protecting the heart and ameliorating lung damage from the disease.Libby and Ridker cautioned that patients who take such life-saving medications should stay on them or at least have a careful discussion with their cardiologists. This is because these drugs have clear and well-established benefits in hypertension and certain forms of heart disease, while their propensity to make humans more susceptible to SARS-CoV-2 remains speculative for the time being.But what remains speculative today will crystalize in the weeks and months to come, Ridker and Libby said, because the science is moving forward rapidly, with new papers coming out daily and a growing pool of patients to draw observations from. Team at Harvard plans to launch clinical trial in fall Health Services director Giang Nguyen talks about adding remote services and new resources for Harvard community Hope for managing hospital admissions of COVID-19 cases “In 12 to 18 months we’re going to have a great deal of information, but right now our job is to, number one, keep people from getting COVID-19 by strict adherence to now-familiar containment measures,” Libby said. “Then, we need to get people who get the disease through this acute phase.”The need for rigorous randomized trials done quickly and effectively is acute, they said. Until the evidence from these trials begins to coalesce, clinicians will have to navigate the uncharted territory of delivering cardiac care in the time of pandemic with caution but also with resolve.“We don’t have the comfort of our usual databases, so we have to rely on our clinical skills and judgment. But we have to do so in all humility because often data don’t bear out our logical preconceptions,” Libby said. “Yet, we must act.” New projections suggest social-distancing measures in state may be flattening the curve
24SHARESShareShareSharePrintMailGooglePinterestDiggRedditStumbleuponDeliciousBufferTumblr Mobile cameras are vital for businesses looking to engage millennials, and banking is the top industry in which that age group wants more mobile-capture functionality (40%), according to findings from new survey.The study, “Millennial – The Next Mobile Disruptors,” from San Diego-based mobile payment provider Mitek and Utica, N.Y.-based Zogby Analytics, examined the mobile preferences of more than 1,000 consumers ages 18 to 34. It revealed millennials want to digitize all commerce through their mobile camera. continue reading »
EC Girls 92Greensburg 91 Next up for the AquaTrojans is a trip to Lawrenceburg to visit the Tigers of Lawrenceburg and the Twisters of Oldenburg Academy. Individual winners include:Kyra Hall 200 Free, 100 BackTyler Ketcham 200 FreeJackson Ketcham 200 IM, 100 BackCaroline Walters 50 FreestyleMatthew Badinghaus 50 Free, 100 FreeAly Bailey 1 meter divingJohn Crawley 1 meter divingMackenzie Schantz 100 Fly, 500 FreeNick Weber 100 Fly, 500 FreeEmily Hafertepen 100 BreastRay Krider 100 Breast EC Boys 138Greensburg 28 EC Boys won all 3 relays. GO AquaTrojans!!! Exciting night for the AquaTrojans against the Greensburg Pirates on Tuesday in St. Leon. EC won a very close meet on the Girls side with the Boys also taking home the victory. The scores were: Courtesy of AquaTrojans Coach Brandon Loveless.